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An early publication in the Annals of the New York Academy of Sciences documenting the original discovery of telomere shortening as a primary mechanism of cellular senescence and its relevance to human aging biology provides the foundational framework for understanding why telomerase-activating compounds attract longevity research attention

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Molecular docking (MOE), alanine-substituted peptide library screening, and surface plasmon resonance (SPR) revealed that BPC157 binds FBXO22 through its proline residue at position 3 (P3)

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